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Impotence - Caused by Cholesterol Drugs

Cholesterol is a lipid of steroids and alcohol found in the cell membranes of all tissues that is transported in blood plasma. Abnormally high cholesterol levels and abnormal proportions of LDL ("bad cholesterol") and HDL ("good cholesterol") are associated with cardiovascular disease by causing artery deposits (atherosclerosis). This

disease process leads to heart attack, stroke, and peripheral vascular disease.  Cholesterol drugs aim to reduce the production of LDL in the liver, but in the process they can have an adverse effect on sexual health.

Unexpected Nightmares

The liver produces LDL in order to support hormonal (steroid) production. In each hormonal conversion, the liver must provide various enzymes for the host organs, such as P450, to do their jobs. Cholesterol drugs reduce the liver steroid enzymes associated with cholesterol-hormone conversion.  In the process of trying to lower LDL by inhibiting the liver steroid enzymes, the prescription drugs inadvertently also inhibit the enzymes that are involved in the production of androgen hormones such as DHEA, androstenedione and testosterone. With a lack of androgen hormones, your penis can shrink or become limp.

Most Statin drugs prescribed for lowering cholesterol on the market are effective in slowing the progression of atherosclerosis. Many clinical researchers have used ultrasound technology that measures the pathological thickening of the aging blood vessel walls for patients taking Statin drugs. It has shown little effect on regression. It’s not surprising, giving the nature of chemical drugs and the challenge of fixing the symptoms. It’s difficult for a chemical compound to both relieve the symptom and eradicate the cause of the problem. [1] [2] [3] [4] [5]

Life is already full of tough choices. There is no need for men to be forced to choose either their heart or penis. There are many natural remedies such as life style, diet changes, and other means to reduce LDH naturally. It is also wise to consult your physicians if you are starting a new approach to improve your health.

Use fish oil (3000 mg Omega-3 to increase prostaglandin E-3), Vitamin B-3 (200-300 mg), and Vitamin B-5 (30-50 mg) are good supplements along with cholesterol drugs. In addition to supplementation, cholesterol problems can be addressed effectively with exercise and a healthy diet.  Increase your intake of fruits and vegetables by 100-300%.

Don’t Forget to Take CoQ10 along with Red Yeast Rice

It is recommended to take CoQ10, for those using a Red Yeast Rice supplement to manage their cholesterol level. Research has shown that Red Yeast Rice containing natural lovastatin inhibits cholesterol synthesis by deactivating HMG-CoA reductase. [6] As a result, circulating cholesterol and LDL-cholesterol are both lowered. It can also reduce CoQ10 levels in the body up to 40%. [7] [8] [9]

Natural Solution

Erectile restorative herbs such as Cuscuta, Butea Superba, Pyrola, Cynomorium, and Epimedium complement well with Omega-3 and CoQ10 to help restore back erection power and prevent further damages to penile blood vessels. Men can lower their cholesterol level and stop LDL drugs from damaging the liver steroid enzymes involved in the production of essential hormones, e.g., DHEA, androstenedione and testosterone. With proper diet, exercise and the right doctor consultation, men can see an improvement in cholesterol levels and erectile strength. 


  1. ^Crouse JR, III, Raichlen JS, Riley WA, et al. Effect of rosuvastatin on progression of carotid intima-media thickness in low-risk individuals with subclinical atherosclerosis: the METEOR Trial. JAMA. 2007 Mar 28;297(12):1344-53.
  2. ^de Groot E, Jukema JW, Montauban van Swijndregt AD, et al. B-mode ultrasound assessment of pravastatin treatment effect on carotid and femoral artery walls and its correlations with coronary arteriographic findings: a report of the Regression Growth Evaluation Statin Study (REGRESS). Am Coll Cardiol. 1998 Jun;31(7):1561-7.
  3. ^Nissen SE, Nicholls SJ, Sipahi I, et al. Effect of very high-intensity statin therapy on regression of coronary atherosclerosis: the ASTEROID trial. JAMA. 2006 Apr 5;295(13):1556-65.
  4. ^Salonen R, Nyyssonen K, Porkkala E, et al. Kuopio Atherosclerosis Prevention Study (KAPS). A population-based primary preventive trial of the effect of LDL lowering on atherosclerotic progression in carotid and femoral arteries. Circulation. 1995 Oct 1;92(7):1758-64
  5. ^Taylor AJ, Kent SM, Flaherty PJ, Coyle LC, Markwood TT, Vernalis MN. ARBITER: Arterial Biology for the Investigation of the Treatment Effects of Reducing Cholesterol: a randomized trial comparing the effects of atorvastatin and pravastatin on carotid intima medial thickness. Circulation. 2002 Oct 15;106(16):2055-60.
  6. ^Edwards JE, Moore RA (December 2003). "Statins in hypercholesterolaemia: a dose-specific meta-analysis of lipid changes in randomised, double blind trials". BMC Fam Pract 4: 18. doi:10.1186/1471-2296-4-18
  7. ^Sarter, B (2002). "Coenzyme Q10 and cardiovascular disease: a review". The Journal of cardiovascular nursing 16 (4): 9–20.
  8. ^Thibault, A; Samid, D; Tompkins, AC; Figg, WD; Cooper, MR; Hohl, RJ; Trepel, J; Liang, B et al. (1996). "Phase I study of lovastatin, an inhibitor of the mevalonate pathway, in patients with cancer". Clinical cancer research 2 (3): 483–91.
  9. ^Ghirlanda, G; Oradei, A; Manto, A; Lippa, S; Uccioli, L; Caputo, S; Greco, AV; Littarru, GP (1993). "Evidence of plasma CoQ10-lowering effect by HMG-CoA reductase inhibitors: a double-blind, placebo-controlled study". Journal of clinical pharmacology 33 (3): 226–9.

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